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Not Yet RecruitingNCT07346443

Mechanism Study of G Protein-coupled Receptor 81 in Multiple Organ Dysfunction of Heat Stroke

Status
Not Yet Recruiting
Phase
Study type
Observational
Enrollment
100 (estimated)
Sponsor
Second Affiliated Hospital of Wenzhou Medical University · Academic / Other
Sex
All
Age
Healthy volunteers
Not accepted

Summary

Heat stroke is one of the common acute and critical conditions in the ICU, characterized by a high incidence and mortality rate, and there is still a lack of effective and precise treatment methods. The latest research indicates that G protein-coupled receptor 81 (GPR81), as an endogenous receptor of lactic acid, can regulate immune responses by up-regulating the expression of inflammatory factors and the chemotaxis of neutrophils, suggesting that the GPR81 receptor may play a potential key role in heat stroke. The investigators aim to reveal the position and key role of GPR81 in improving multiple organ dysfunction caused by heat stroke, and to clarify its specific molecular mechanism, with the goal of providing new ideas for the prevention and treatment of heat stroke based on lactate levels, a commonly used clinical indicator.

Conditions

Timeline

Start date
2026-03-01
Primary completion
2026-12-31
Completion
2027-01-31
First posted
2026-01-16
Last updated
2026-01-16

Source: ClinicalTrials.gov record NCT07346443. Inclusion in this directory is not an endorsement.